Original Article Long non-coding RNA ZEB2-AS1 promotes
30 août 2019 Original Article. Long non-coding RNA ZEB2-AS1 promotes proliferation and inhibits apoptosis of colon cancer cells via miR-143/bcl-2 axis.
ajtr
lncRNA UCA1 Mediates Resistance to Cisplatin by Regulating the
As shown in Figure 4A miR-143 harbors a complementary binding sequence to UCA1. Figure 2. UCA1 Affects the Proliferation and. Apoptosis of Ovarian Cancer In
S ( )
Original Article Identification of the potential targets of miR-143-3p in
28 févr. 2017 between miRNAs and CRC. MiR-143-3p is a type of miRNA that was recently found to be involved in regulating proliferation and apopto-.
ijcem
Original Article MiR-143-3p suppresses the progression of ovarian
30 mars 2018 down-regulated the expression of transforming growth factor ... Keywords: MiR-143-3p ovarian cancer
ajtr
Exosomal miR-143-3p derived from follicular fluid promotes
noncoding RNA molecules that can regulate target gene expression by exosomal-miR-143-3p on the apoptosis and proliferation of ovarian GCs and then ...
s ?origin=ppub
lncRNA UCA1 Mediates Resistance to Cisplatin by Regulating the
As shown in Figure 4A miR-143 harbors a complementary binding sequence to UCA1. Figure 2. UCA1 Affects the Proliferation and. Apoptosis of Ovarian Cancer In
Exosomes Derived from miR-143-Overexpressing MSCs Inhibit Cell
26 févr. 2019 Figure 5. Exosomal miR-143 Regulates Proliferation Migration
S ( )
Original Article Significant role and mechanism of microRNA-143-3p
30 juin 2019 MiR-143-3p controls TGF-β1-induced cell proliferation and extracellular matrix production in airway sm- ooth muscle via negative regulation of ...
ajtr
MicroRNA-143 targets MAPK3 to regulate the proliferation and bone
ORIGINAL ARTICLE. MicroRNA-143 targets MAPK3 to regulate the proliferation and bone metastasis of human breast cancer cells. Yiqun Du12
miR-143 interferes with ERK5 signaling and abrogates prostate
12 nov. 2009 Since miR-143 was found to be down-regulated in prostate cancer cells ... Decreased proliferation and increased apoptosis in.