Received 30 May1990/Accepted 20 August 1990 The immunopathology of Chagas' disease was studied in the experimental model of chronic infection in C57BL/lOJTormice Sublethal infection with Trypanosoma cruzi, Ystrain, induced specific antibodies anda delayed hypersensitivity response to parasite antigens Micedeveloped chronic chagasic
Therapeutics, Glasgow-Scotland, with Professor John L Reid (1989-1990) 2 Cellular and Molecular Mechanisms of American Trypanosomiasis (Chagas Disease) and Development of Antichagastic Agent: Parasite (T Cruzi) Binding, Infectivity, and Multiplication in Rat Heart Myoblast and Effects of Polyamine Synthesis Inhibitors
286286 Serological Diagnosis of Chagas Disease Ł ES Umezawa, JF Silveira high sensitivity, specificity, and positive and nega-tive predictive values in previous studies (Moncayo & Luquetti 1990, Levin et al 1991) Two non re-petitive antigens A13 (Paranhos et al 1990) and 1F8 (Gonzalez et al 1985) were also included in this evaluation
1990-99 Sound Director and Composer in Residence Paulo C Chagas Curriculum Vitae 2 of 33 [accessed August 1, 2013] Chagas, Paulo C 2008 “Composition in
It was only in 1990 that the results of a national entomological survey became known (Corredor et al 1990), and in 1995 data were published to show the national prevalence of infection in blood do-nors in the country (Guhl et al 1995) These re-ports represented major steps in medical surveil-lance, laying the basis for current control activi-
In 1990 and 1994, mass vaccination was performed by using a vaccine consisting of a serogroup B outer membrane vesicle (B:4:P1 15) and a serogroup C polysaccharide During 1993–1994, a total of 4 C:4 N meningitidis isolates were identifi ed in samples from patients in Rio de Janeiro State (RJ) The presence of class3-PorB in these strains
Carvalho et al , 1990), chaperonin (Giambiagi-de Marval et al , 1993; Fernandes et al , 2005) and small HSP (Pérez-Morales et al , 2009) families have been described Of the HSP100 family, the HSP104 gene structure has been described in T brucei (Gottesman et al , 1990), Leishmania major
[PDF]
Impaired autonomic control of heart interval changes to
interval variation (Junqueira, 1990) in subjects with appa-rent indeterminate form of Chagas’ disease in comparison with healthy controls, which may contribute to the under-standing of the pathophysiology of this important and intriguing condition 2 Subjects and methods 2 1 Subjects A group of 36 consecutively recruited outpatients (15
[PDF]
Parasympathetic Impairment in the Preclinical Stage of
in Chagas’ disease appears to be a primary phenomenon of varying intensity without a strict relation of cause and effect to the progressive contractile disturbance2 In the early studies on Chagas heart disease, the autonomic disturbances were already considered to be the cause of the progressive mechanical alterations of the heart Similar to the mechanism of the digestive disease,
[PDF]
SEROPREVALENCE OF CHAGAS DISEASE IN SCHOOLCHILDREN
volunteers, indicated a Chagas disease infection prevalence of 18 0 with rare cases of positivity observed in children under 10 years of age (0 17 ), suggesting interruption of the vectorial transmission of the disease and success in vector control actions23 The frequent finding of
[PDF]
Prevalence of Chagas disease in Brazil: A systematic
The pooled estimate of Chagas disease prevalence across studies for the entire period was 4 2 (95 CI: 3 1–5 7), ranging from 4 4 (95 CI: 2 3–8 3) in the 1980s to 2 4 (95 CI: 1 5–3 8) after
[PDF]
Urban transmission of Chagas disease in Cochabamba, Bolivia
Mem Inst Oswaldo Cruz, Rio de Janeiro, Vol 103(5): 423-430, August 2008 423 online memorias ioc fiocruz br Urban transmission of Chagas disease in
[PDF]
Rio de Janeiro, Vol 94, Suppl I: 413-415, 1999 413
It was only in 1990 that the results of a national entomological survey became known (Corredor et al 1990), and in 1995 data were published to show the national prevalence of infection in blood do-nors in the country (Guhl et al 1995) These re-ports represented major steps in medical surveil-lance, laying the basis for current control activi-
[PDF]
World Bank Document
Objectives: disesases (Chagas' disease, schistosomiasis and leielbaniasis) by reducing the prevalence and intensity of human infection in the population at risk of Brazil's Northeast, and to improve health status and productivity An additional o1jective is to support MOB activities to reduce
[PDF]
Polymerase Chain Reaction Detection: New Insights into the
been questioned, due to the frequency of infections with other trypanosomatids that circulate in the same geographic area of T cruzi and are respon-sible for cross-antigenicity and false positive re-sults (AW Ferreira 1992 Tests for Chagas disease serodiagnosis: a review, p 179-193 In S Wendel, Z Brener, ME Camargo & A Rassi (eds) Chagas Disease (American Trypanosomiasis): its Impact on
[PDF]
Review of the application of the traditional Chinese
by fungi Chagas disease is a common epidemic disease and second in frequency only to malaria in Latin America It is caused by Trypanosoma cruzi Schinella et al (2002) screened 18 plant species and two types of bacterium to determine their inhibitory effects on T cruzi Methanol extracts of the aerial part of R sceleratus and the root of